Researchers have uncovered a link between motor neurons' inability to repair oxidative genome damage in ALS, suggesting that DNA ligase-targeted therapies may prevent or slowdown disease progression. The defect found was with DNA ligase III, an important enzyme for connecting single-stranded DNA breaks in mature neurons that, once worn out, are no longer capable of new growth or being replenished in the body.
from Top Health News -- ScienceDaily https://ift.tt/2qnnBOJ
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